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Dihexa demonstrates unusually high oral bioavailability for a peptide compound, estimated at 40-60% in animal models compared to the typical 5% or less for most peptides
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Oxidative stress therefore accelerates telomere shortening beyond the end-replication problem alone, creating a feed-forward loop in which mitochondrial dysfunction generates reactive oxygen species (ROS) that damage telomeres, triggering senescence and further mitochondrial dysfunction
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Long-term considerations Most safety data on AOD 9604 comes from studies lasting 12 weeks or less
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