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It is released in response to food intake and functions by stimulating insulin release in a glucose-dependent manner, inhibiting glucagon secretion, and slowing gastric emptying, thus contributing to the regulation of blood glucose levels [1]
The restoration of neuron density with GABA treatment underscores its neuroprotective properties, suggesting a protective effect against neuronal loss
We next assayed cAMP-dependent protein kinase (PKA) (27, 35), ERK (36), and eNOS (37) activations as putative downstream targets of GLP-1(2836)induced cAMP and/or molecules known to be involved in GLP-1mediated cytoprotection (21)
Furthermore, the review explores novel drug delivery strategies, such as microsphere encapsulation, injectable hydrogels, Fc-fusion, and oral small-molecule formulations, aimed at achieving sustained and controlled release to reduce dosing frequency and improve patient compliance
It means the evidence base is still developing
