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Therefore, hyperhomocysteinemia directly damages neuronal sheath or indirectly through the induction of abnormal immune response (Mititelu et al
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Despite the complexity of TMAOs role in atherosclerosis, studies have indicated that managing TMAO through nutrition, medication, or microbiome modification may help lessen its effects and even reverse its contribution to the advancement of the disease [73, 76]
"An enzyme from rat liver catalysing conjugations with glutathione"
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