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Bull Exp Biol Med 167:641644

Yap deletion in isolated hepatocytes resulted in maintenance of ALB and HNF4 expression (Sun et al., 2019)

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In this context, the present study was conducted to systematically investigate potential targets and pathogenic pathways of DEHP-induced BRCA specific to Homo sapiens via network toxicology and molecular docking strategies

Therefore, one may extrapolate that these mechanisms could also be recapitulated in PD or other neurodegenerative diseases, and that abnormal mitochondrial glycome could trigger the acceleration of mitochondrial dysfunction and neuronal death