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60 In contrast, the long-acting enzyme resistant form of GLP-2, namely (Gly2)GLP-2, did not provide benefit on bone strength in a mouse model of bone fragility, 61 despite promoting the expression of bone matrix genes and reducing osteoclast number in the in vitro setting
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After depletion of the endogenous protein, cells with intact chromatin bridges expressing CB exhibited nuclear chromatin shape-deformation, relatively high frequency of Sun2 nuclear lines and low frequency of loose DNA bridges, similar to GFP controls (Fig
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