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10.1016/j.neulet.2014.04.020 Neurosci
Constitutive activation of NRF2, mainly via loss-of-function mutations in the CUL3-adapter KEAP1 or gain-of-function mutations in NFE2L2 , is thought to provide a fitness advantage to tumor cells in confronting metabolic/oxidative stress resulting from dysregulated growth (DeNicola et al, 2011
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Semin Hematol 1990
Subtle changes such as persistent fatigue, abnormal bleeding, or gastrointestinal symptoms can provide important early signals that adjustments may be needed
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