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In the initial stages of treatment with lithium, hypercalcaemia occurs as a result of lithiums inhibition of the calcium-sensing receptor (CaSR) and glycogen synthase kinase-3 (GSK-3)
10.3389/fendo.2025.1579477 62
In vascular endothelium, GLP-1R stimulation activates both cAMP/PKA and PI3K/Akt signaling cascades, leading to phosphorylation of endothelial nitric oxide synthase (eNOS) and increased nitric oxide production [4]
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