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Inhibitors of ACSL4 reduce this vulnerability, preserving membrane integrity and protecting lung cells from ferroptotic injury ( Table 1 )
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(2020) specifically addressed NSAID-induced gut damage a clinically relevant injury model and documented that BPC-157 administration stabilized the claudin-4 and occludin tight junction proteins that are degraded by NSAID exposure, providing a mechanistic explanation for the peptides permeability-protective effects
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