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6j), confirming the dependency of GSH-deficient FoB on glycolysis
SCFAs activate GPCRs (GPR43/41) and inhibit histone deacetylases (HDAC), promoting regulatory T-cell (Treg) differentiation while suppressing Th17 cell polarization within the joint, thereby restoring immune homeostasis and reducing pro-inflammatory cytokine release
Evrard, S
The crosstalk between distinct RCDs is well documented and can be interpreted through the lens of the cell death-governing intracellular system with multiple redundant backup pathways [20, 142]
On the mechanism of multiple lysine methylation by the human mixed lineage leukemia protein-1 (mll1) core complex
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