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118 intraperitoneally injected CL82198, the MMP13 inhibitor in a murine model of injury-induced knee OA, which effectively decelerated OA progression, increased extracellular matrix production, and inhibited chondrocyte apoptosis

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H3K36me3 levels correlated with the ability of 1C modulators to promote reprogramming efficiency: MAT2Ai reduced the amount of H3K36me3, while SAM enhanced it (Fig

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