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As a result, glutathione is at the forefront of mitigating quinone-derived toxicity, by enzymatically reducing redox-generated H 2 O 2 to harmless H 2 O via glutathione peroxidase, or by direct reaction and detoxification of the quinone electrophile (Fig
Antioxidant and anti-inflammatory signalling: Published research has shown interactions with antioxidant defence systems and inflammatory signalling pathways in laboratory models, including modulation of pro-inflammatory cytokine activity and upregulation of antioxidant enzyme expression
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Moreover, we also observed a significant reduction in the weight of the cisplatin + RSL3 tumours when compared with the vehicle-treated group at the end point (Fig
Oral availability : BPC-157 may have oral activity (unusual for peptides)
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