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GNE inhibition results in hyposialylation of IGF-1R Since the key function of GNE is epimerization and phosphorylation of precursors in the sialic acid biosynthetic pathway, absence of functional GNE would cause hyposialylation and affect sialylation of cell surface receptors 15,30

In contrast, recent meta-analyses suggest that GLP-1 RA use does not increase the risk of pancreatitis and/or pancreatic cancer, compared to placebo and/or other treatments [101,102,103]

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