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Carcinogenesis 12 , 14711475 (1991)

FOXO4-DRI enters senescent cells and competitively binds to p53 through its modified peptide sequence, displacing endogenous FOXO4 and freeing p53 to activate cell death pathways including BAX and PUMA

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4e) and was incapable of phenotypically rescuing nhr-49(nr2041) mutant defects with respect to lipid deposition and fecundity (Fig