Further, to infer the mechanism of GSH mediated osmotic protection, they followed a comparative proteomic approach which revealed upregulation of several glycolytic enzymes in GSH-supplemented cells during osmotic challenge
Department of Genetics and Clinical Immunology, National Institute of Tuberculosis and Lung Diseases, Warsaw, Poland Abstract Reactive oxygen and nitrogen species can be generated endogenously (by mitochondria, peroxisomes, and phagocytic cells) and exogenously (by pollutions, UV exposure, xenobiotic compounds, and cigarette smoke)
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