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Neuronal membranes, abundant in polyunsaturated fatty acids, are highly susceptible to ROS and experience lipid peroxidation [4]
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Although -oxidation is impaired in the setting of VPA toxicity, this metabolite has a prolonged elimination half-life and could be responsible for the prolonged coma that is sometimes observed despite the normalization in plasma VPA concentrations [17, 18]
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Patients should inform their healthcare provider of all medications and supplements they are taking to minimize the risk of adverse interactions, and healthcare professionals should evaluate each regimen prior to initiating glutathione therapy
