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QTc prolongation is caused by drugs used to treat diabetes, schizophrenia, and other psychiatric conditions [14]
Inhibiting the activity of system Xc affects GSH synthesis by inhibiting cystine absorption, leading to decreased GPX activity, reduced cellular antioxidant capacity, accumulation of lipid ROS, and ultimately oxidative damage and ferroptosis
Once ingested, it is rapidly deacetylated by the body to yield free cysteine, completely bypassing the metabolic bottlenecks that often plague chronically ill patients
Expression of human NAT1 has been detected in adult liver, bladder, digestive system, blood cells, placenta, skin, skeletal muscles, gingiva (Dupret & Lima, 2005), mammary tissue, prostate, and lung by a number of methods (Sim et al., 2008)
[PMID: 6373044] PubMed record related to this compound for laboratory literature review
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