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In addition, exploring molecular pathways underlying the crosstalk among ferroptosis, ocular immunity, and neuroinflammation may clarify the precise role of microglial activation/Mller cell dysfunction in retinal degeneration

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[4] [5] It is encoded by the codons CAA and CAG

Spatial analyses have shown that necrotic regions display significantly greater local propagation activity than apoptotic regions, indicating that the mode of death execution itself may govern the transition from isolated cell death to multicellular ferroptotic contagion