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SARS-CoV-2 infects type II pneumocytes and disrupts the cellular Fe-RH with increased ROS release causing severe OxS
Proapoptotic activity of new glutathione S-transferase inhibitors
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Concurrently, moderate levels of ROS serve as critical secondary messengers that activate key pro-survival and proliferative signaling pathways including MAPK, PI3K/AKT, NF-B, and HIF-1
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