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GLP-1 delays gastric emptying via stimulating inhibitory nitrergic myenteric neurons [18] and activating vagal afferents [19], leading to reduced gastrointestinal motility [20]
Participants were generally prescribed GLP-1RAs for non-alcohol-related indications and were not specifically recruited for AUD treatment
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Among them, 7 are GLP-1R agonists, including 3 biosimilars of Semaglutide
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