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Thus, the action of LAC on BDNF levels and the rapid regulation of the glutamatergic system is in line with its antidepressant activity
(49) have confirmed that in vitro treatment with UA can reduce the expression levels of fibrosis markers including vimentin, discoidin domain receptor tyrosine kinase 2, tensin and Alpha-smooth muscle actin in myocardial tissues, and inhibit the transition of cardiac fibroblasts to a fibrotic state through nuclear factor erythroid 2-related factor 2 signaling pathway
Glutathione is generally considered safe when taken in appropriately low doses
V., & Contel, M
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