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Laboratory studies in animals indicate that GLP-1 RAs target hallmarks of brain aging and may activate neuroprotective pathways that promote cellular waste removal, reduce neuroinflammation, promote mitochondrial health and regeneration which may alleviate symptoms of cognitive decline in models of Parkinsons and Alzheimers diseases
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Understanding their mechanism of action provides a framework for optimizing efficacy, minimizing side effects, and expanding applications beyond diabetes to include other areas such as the treatment of obesity, cardiovascular diseases, non-alcoholic fatty liver disease (NAFLD), and even neurodegeneration
12 TP-H then acts as a catalytic antioxidant scavenger
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