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The release and excessive accumulation of collagen fibers caused by overactivated fibroblasts, which are marked with fibroblast activation protein (FAP), lead to pathological changes in the extracellular matrix (ECM) and persistent inflammation, resulting in a vicious cycle of cellular activity and extracellular fibrosis 3,4,5,6,7

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When BPC-157 is stored beyond its viable period or under poor conditions, several degradative processes may occur: Hydrolysis of peptide bonds leading to breakdown of the chain and loss of activity

FEBS Letters, 238(2):343346