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To dissect the actions of AR signaling in a range of cell types that contribute to the process of fracture healing, we generated genetically engineered mice in which the AR gene was specifically ablated in periosteal cells (AR -/Y ;Prrx1::Cre mice) or chondrocytes (AR -/Y ;Col-2::Cre mice), and created a closed transverse femoral fracture to compare the bone healing with that in AR flox/Y mice

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