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Our results demonstrate for the first time that HSV-1 acute infection triggers H2AX formation and accumulation in primary neurons, indicating that in these cells the virus is able to induce DNA lesions and DDR events

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In neuronal cell cultures, GHK-Cu demonstrates remarkable neuroprotective effects, reducing oxidative damage by 55% and enhancing cell survival under stress conditions by 40% compared to control administrations

The identification of predictive biomarkers could further optimize these combination strategies (114, 115)