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Various structural alterations were made at the N-terminal such as substitution of D-norleucine for L-norleucine, N-acetylation of norleucine, and replacement of norleucine with GABA in order to improve overall stability by reducing susceptibility to aminopeptidases
Minimal proliferative capacity in adults
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The treatment of PFIC has long been based on the use of drugs, such as ursodeoxycholic acid (UDCA), rifampicin, and cholestyramine, or surgical biliary diversion (SBD), aimed at reducing symptoms, especially pruritus [13]
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