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Early studies focused on its apparent stability within gastric environments and its interactions with pathways associated with mucosal protection and gastrointestinal integrity

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Several papers describe upregulation of the vascular endothelial growth factor receptor 2 ( VEGFR2 ) pathway and downstream nitric oxide signalling the so-called VEGFR2-Akt-eNOS axis which researchers hypothesise underlies its reported influence on wound healing and blood-flow restoration at an injury site

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