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If medically necessary for a patient, healthcare providers can prescribe compounded medications with similar mechanisms of action to gain greater control of the patient-specific disease process being treated

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Upon ligand binding, TR II phosphorylates and activates TR I, forming a heteromeric receptor complex that initiates downstream signaling through both SMAD-dependent and SMAD-independent pathways, such as ERK, p38, JNK, and PI3K-Akt cascades [33, 36, 39]

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