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By activating GLP-1 and GIP receptors, it enhances insulin secretion (when glucose is present) and suppresses appetite, similar to existing incretin therapies [7] [8]
Additional analysis explored the interaction between BPC 157 and several neurotransmitter and signaling systems, including dopaminergic, nitric oxide, prostaglandin, and somatosensory pathways
We avoid using tertiary references
no human safety data beyond preclinical exposures
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