The overexpression of DYRK1A in T21 brains may contribute directly to early-onset neurofibrillary degeneration through tau hyperphosphorylation and indirectly through the phosphorylation of alternative splicing factors (Wegiel et al
Double anthracycline (mitoxantrone + idarubicin) therapy with cyclosporin a (CSA) to block multidrug resistance (MDR) as salvage therapy for relapsed or refractory acute myelogenous leukemia (R-AML)
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Inhibition of ALDH1A1 activity decreases expression of drug transporters and reduces chemotherapy resistance in ovarian cancer cell lines
