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Instead, these organs employ an amino acid transporter to absorb glycine, cysteine, and glutamate for intracellular de novo GSH synthesis

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Clinicians and researchers have recently emphasized the need to consider frailty and sarcopenicobese phenotypes [132]: frail individuals with high cardiometabolic risk may benefit, whereas anorexic or malnourished phenotypes are vulnerable to excessive weight loss or dehydration [133]

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