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The in silico process can work in a reversed engineered fashion by interrogating databases with biomarkers of harm or transcriptional changes across a spectrum of related CVD landscape (e.g., atherosclerosis due to high-fat, or hypertension) to derive a subset of overlapping markers/genes that point to perhaps known or novel targets (Graphical Abstract), and analogous to a drug-repurposing algorithm.[198] Abundant publicly available databases (clinical, animal models, cells, etc.) on DIVI provide ample starting material for understanding how drugs of a specific class may initiate DIVI