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Direct answer: Oxidative stress is not a disease by itself
Studies show that it has a similar mechanism of action as AOD9604
For instance, by quantitatively measuring oxidative damage markers (e.g., 8-OHdG) or core inflammatory cytokines (e.g., IL-8, TNF-$\alpha$) in seminal plasma, patients can be precisely subtyped into high oxidative stress or high inflammation phenotypes
Salk Institute researchers are burrowing down into the mechanistic details behind how GLP-1 drugs promote viability and stress resistance in pancreatic beta cells
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