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Given that recent genomic profiling indicates SKOV-3 may not strictly represent HGSOC, further investigations using diverse and clinically representative HGSOC cell lines are required to fully validate these ROS-driven metastatic mechanisms

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Low levels of ROS can activate host cells in the tumor microenvironment, and promote glucose metabolism in tumors to maintain the high energy consumption of tumors by inducing mitochondrial autophagy, altering key enzymes and genes related glucose metabolism and activating signal pathways

Zhang et al., 2024a)