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Stress exposure, particularly early-life adversity, may dysregulate endogenous ALC signaling and moderate treatment response (Figure 4)

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Because many previous reports demonstrated that hypertension alone induces a shift from fatty acid oxidation toward increased glucose utilization in cardiac muscle, hypertension complicated with HFpEF might have additive effects on energy metabolic failure in heart

10.1016/j.nut.2019.110664 Burnett JR, Hooper AJ, Hegele RA: Abetalipoproteinemia