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Abstract Non-alcoholic steatohepatitis (NASH) is emerging as a major cause of hepatocellular carcinoma (HCC), however, it is not resolved if compounds in late-stage clinical development for NASH may have additional therapeutic benefits in NASH-driven HCC (NASH-HCC)
Necrosis, typically resulting from severe injury, manifests as uncontrolled cell death with cellular content leakage, whereas ferroptosis proceeds as a regulated process driven by oxidative stress and iron overload, primarily through ROS-induced lipid damage (14)
10.2174/1381612824666180614082950 56 KatoN.KawaiT.MaedaH.NagaoT.MurakamiH.MiyazawaK.et al (1995)
The skin cells have decreased proliferative ability, as they cannot re-enter the mitotic circle and eventually, they begin the process of apoptosis [8, 9]
