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These findings are compatible with the previous studies, which revealed iron overload-mediated free radicals stress can result in cellular and mitochondrial dysfunction 58,59,60

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(6) In activated macrophages, GHK-Cu apparently lowered intracellular reactive oxygen species and restored superoxide dismutase activity toward control values

The influence of MTHFR on psychiatric diseases is mainly through reduction of MTHFR activity which results in elevation of homocysteine, reduction of DNA methylation-dependent methyl donor, finally induces hypomethylation, and then active disease-related genes