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This modification orchestrates TME immunosuppression through dual mechanisms by suppressing effector immunity by impairing cytosolic DNA sensing via cGAS activity attenuation and amplifying TGF- signaling cascades in cytotoxic lymphocytes, while simultaneously enabling immune checkpoints by upregulating PD-L1 expression on antigen-presenting cells and stabilizing immunosuppressive Treg and TAM phenotypes (63)

What It Treats

Interestingly, we found in our study that GPX1 is not capable of oxidative stress in all cases

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