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Pharmacokinetic and Biochemical Profile GHK-Cu characterization in preclinical research reveals important properties for experimental design: Stability and Distribution: High copper complex stability (Ka ~10) Resistant to peptidase degradation when copper-coordinated Tissue penetration enhanced by small size and lipophilicity Plasma protein binding minimal for free peptide Cellular uptake observed in various cell types Biological Half-Life: Plasma half-life: Short as free peptide (minutes to hours) Tissue retention: Extended due to cellular uptake and binding Copper delivery to cells: Sustained over extended periods Biological effects: Persist beyond plasma clearance suggesting intracellular actions Copper Delivery Function: Facilitates copper transport into cells Modulates cellular copper distribution Activates copper-dependent enzymes Regulates copper-responsive gene expression These characteristics inform research protocol design

This was still far below the results seen with surgery

Nevertheless, several studies addressing the issue of post-stroke dementia reported a persistent and steeper rate of cognitive decline after stroke over a long-term follow-up of 612 years, even after correcting for its pre-stroke rate, leading to an independent increase in mortality (Zheng et al., 2019)