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doi: 10.3390/pharmaceutics15122717 16 SuWLiYYangGZhaoYZhouXLiuG

Hyperglycemia acts as both an initiator of oxidative stress and a co-promoter of pathogenic molecular pathways, and cross-talk among these pathways induces ROS amplification via positive feedback, exacerbating oxidative damage, BRB impairment, iron dyshomeostasis and DR progression (157, 158)

Consistently, overexpression of hHPN WT in hepsin / mice resulted in a significant increase in APAP tolerance, with survival rates exceeding 80% even after 80 h (Fig

L-Buthionine sulfoximine (BSO) is a specific -glutamylcysteine synthetase inhibitor that blocks the rate-limiting step of glutathionine (GSH) biosynthesis and in doing so depletes the intracellular GSH pool in both cultured cells and in whole animals [10]