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In TNBC, impairment of apoptotic signaling enables tumor cells to evade immune-mediated clearance, promoting sustained proliferation, metastasis, and resistance to therapy [42]
However, simpler experiments in intact brain tissue (e.g., immunohistochemistry), brain slices, cultured brain cells, and mitochondria have also made important contributions to the understanding of details, mechanisms, and functional consequences of glutamate/GABA biosynthesis and degradation
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