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These data demonstrate that high OxPhos signalling is associated with 5FU resistance and suggest that enhanced mitochondrial metabolism promotes treatment failure clinically
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Stress conditions activate mast cells to release prestored and newly synthesized inflammatory mediators and induce increased blood-brain barrier permeability, recruitment of immune and inflammatory cells into the brain and neuroinflammation
Downtime Minimal: Setelah perawatan, waktu pemulihan sangat minim sehingga Anda bisa kembali beraktivitas tanpa gangguan
doi: 10.1016/j.ebiom.2020.102793 166 LiYLHungWC
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