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In the brain, SELENOP is primarily expressed in astrocytes, but neurons have also been identified as a source of endogenous SELENOP through the entire brain (Steinbrenner et al., 2006
Liraglutide Protects Neurite Outgrowth of Cortical Neurons Under Oxidative Stress though Activating the Wnt Pathway
Nevertheless, GLP-1 presented a rather ephemeral circulatory half-life, lasting a mere 12 min, due to the rapid enzymatic degradation by dipeptidyl peptidase IV (DPP-4), which markedly limits its biological efficacy [3,4,5]
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