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Collagen and Extracellular Matrix GHK-Cu stimulates collagen synthesis through multiple mechanisms

These tumor cells were used to model disease-specific phenotypes

a , Cell death quantification (% trypan blue + ) of vehicle and DFO-treated (48 h) Huh7 cells under glucose starvation

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The compound is endogenously released at sites of tissue injury through breakdown of SPARC and type I collagen meaning its biology connects directly to repair signaling rather than acting as an exogenous-only tool compound