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for this reason, GSH is being investigated as a potential therapeutic method, as it is the main protective mechanism against elevated oxidative stress
It can be used to combat issues with fat metabolism
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Bid truncation led to ROS-dependent mitochondrial outer membrane permeabilization (MOMP) and cytochrome c release in MV4-11 and U937 cells at 3 and 6 h, respectively (F ig
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